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anti cd166 apc  (Miltenyi Biotec)


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    Structured Review

    Miltenyi Biotec anti cd166 apc
    Anti Cd166 Apc, supplied by Miltenyi Biotec, used in various techniques. Bioz Stars score: 94/100, based on 16 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Average 94 stars, based on 16 article reviews
    anti cd166 apc - by Bioz Stars, 2026-03
    94/100 stars

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    A : Fluorescent staining of the outer layers of the retina showing comparable zpr1 (Arr3a) staining in the photoreceptor outer segments and pedicles in the outer plexiform layer, but reduced and disrupted synaptophysin (syp) staining, protrusion of photoreceptor cell bodies in the outer plexiform layer (arrowheads), and disorganization of horizontal cells lining the top of the outer nuclear layer (asterisks) suggesting defects to the ribbon synapses (40X magnification, scale bars: 20 µm for main panels, 10 µm insets). B : Quantification of synaptophysin intensity in the outer plexiform layer showing a significant reduction in MZKOs (**p = 0.0069). C : Fluorescent staining of transverse cryosections <t>with</t> <t>zn-8</t> and DAPI showing an overall disruption in morphology of the retina with evidence of retinal ganglion cell axon defasciculation in the optic nerves at the chiasm (10X magnification, scale bars: 100 µm for main panels, 20 µm for chiasm insets).
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    Miltenyi Biotec anti cd166 apc
    A : Fluorescent staining of the outer layers of the retina showing comparable zpr1 (Arr3a) staining in the photoreceptor outer segments and pedicles in the outer plexiform layer, but reduced and disrupted synaptophysin (syp) staining, protrusion of photoreceptor cell bodies in the outer plexiform layer (arrowheads), and disorganization of horizontal cells lining the top of the outer nuclear layer (asterisks) suggesting defects to the ribbon synapses (40X magnification, scale bars: 20 µm for main panels, 10 µm insets). B : Quantification of synaptophysin intensity in the outer plexiform layer showing a significant reduction in MZKOs (**p = 0.0069). C : Fluorescent staining of transverse cryosections <t>with</t> <t>zn-8</t> and DAPI showing an overall disruption in morphology of the retina with evidence of retinal ganglion cell axon defasciculation in the optic nerves at the chiasm (10X magnification, scale bars: 100 µm for main panels, 20 µm for chiasm insets).
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    Miltenyi Biotec anti cd166 pe alcam
    A : Fluorescent staining of the outer layers of the retina showing comparable zpr1 (Arr3a) staining in the photoreceptor outer segments and pedicles in the outer plexiform layer, but reduced and disrupted synaptophysin (syp) staining, protrusion of photoreceptor cell bodies in the outer plexiform layer (arrowheads), and disorganization of horizontal cells lining the top of the outer nuclear layer (asterisks) suggesting defects to the ribbon synapses (40X magnification, scale bars: 20 µm for main panels, 10 µm insets). B : Quantification of synaptophysin intensity in the outer plexiform layer showing a significant reduction in MZKOs (**p = 0.0069). C : Fluorescent staining of transverse cryosections <t>with</t> <t>zn-8</t> and DAPI showing an overall disruption in morphology of the retina with evidence of retinal ganglion cell axon defasciculation in the optic nerves at the chiasm (10X magnification, scale bars: 100 µm for main panels, 20 µm for chiasm insets).
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    A : Fluorescent staining of the outer layers of the retina showing comparable zpr1 (Arr3a) staining in the photoreceptor outer segments and pedicles in the outer plexiform layer, but reduced and disrupted synaptophysin (syp) staining, protrusion of photoreceptor cell bodies in the outer plexiform layer (arrowheads), and disorganization of horizontal cells lining the top of the outer nuclear layer (asterisks) suggesting defects to the ribbon synapses (40X magnification, scale bars: 20 µm for main panels, 10 µm insets). B : Quantification of synaptophysin intensity in the outer plexiform layer showing a significant reduction in MZKOs (**p = 0.0069). C : Fluorescent staining of transverse cryosections <t>with</t> <t>zn-8</t> and DAPI showing an overall disruption in morphology of the retina with evidence of retinal ganglion cell axon defasciculation in the optic nerves at the chiasm (10X magnification, scale bars: 100 µm for main panels, 20 µm for chiasm insets).
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    Miltenyi Biotec igg1 pe cd166 miltenyi biotec
    A : Fluorescent staining of the outer layers of the retina showing comparable zpr1 (Arr3a) staining in the photoreceptor outer segments and pedicles in the outer plexiform layer, but reduced and disrupted synaptophysin (syp) staining, protrusion of photoreceptor cell bodies in the outer plexiform layer (arrowheads), and disorganization of horizontal cells lining the top of the outer nuclear layer (asterisks) suggesting defects to the ribbon synapses (40X magnification, scale bars: 20 µm for main panels, 10 µm insets). B : Quantification of synaptophysin intensity in the outer plexiform layer showing a significant reduction in MZKOs (**p = 0.0069). C : Fluorescent staining of transverse cryosections <t>with</t> <t>zn-8</t> and DAPI showing an overall disruption in morphology of the retina with evidence of retinal ganglion cell axon defasciculation in the optic nerves at the chiasm (10X magnification, scale bars: 100 µm for main panels, 20 µm for chiasm insets).
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    Thermo Fisher cd166-percp-efluor710 3a6 antibody
    A : Fluorescent staining of the outer layers of the retina showing comparable zpr1 (Arr3a) staining in the photoreceptor outer segments and pedicles in the outer plexiform layer, but reduced and disrupted synaptophysin (syp) staining, protrusion of photoreceptor cell bodies in the outer plexiform layer (arrowheads), and disorganization of horizontal cells lining the top of the outer nuclear layer (asterisks) suggesting defects to the ribbon synapses (40X magnification, scale bars: 20 µm for main panels, 10 µm insets). B : Quantification of synaptophysin intensity in the outer plexiform layer showing a significant reduction in MZKOs (**p = 0.0069). C : Fluorescent staining of transverse cryosections <t>with</t> <t>zn-8</t> and DAPI showing an overall disruption in morphology of the retina with evidence of retinal ganglion cell axon defasciculation in the optic nerves at the chiasm (10X magnification, scale bars: 100 µm for main panels, 20 µm for chiasm insets).
    Cd166 Percp Efluor710 3a6 Antibody, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Miltenyi Biotec musc isolation
    A : Fluorescent staining of the outer layers of the retina showing comparable zpr1 (Arr3a) staining in the photoreceptor outer segments and pedicles in the outer plexiform layer, but reduced and disrupted synaptophysin (syp) staining, protrusion of photoreceptor cell bodies in the outer plexiform layer (arrowheads), and disorganization of horizontal cells lining the top of the outer nuclear layer (asterisks) suggesting defects to the ribbon synapses (40X magnification, scale bars: 20 µm for main panels, 10 µm insets). B : Quantification of synaptophysin intensity in the outer plexiform layer showing a significant reduction in MZKOs (**p = 0.0069). C : Fluorescent staining of transverse cryosections <t>with</t> <t>zn-8</t> and DAPI showing an overall disruption in morphology of the retina with evidence of retinal ganglion cell axon defasciculation in the optic nerves at the chiasm (10X magnification, scale bars: 100 µm for main panels, 20 µm for chiasm insets).
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    Miltenyi Biotec mouse anti human cd166 allophycocyanin
    A : Fluorescent staining of the outer layers of the retina showing comparable zpr1 (Arr3a) staining in the photoreceptor outer segments and pedicles in the outer plexiform layer, but reduced and disrupted synaptophysin (syp) staining, protrusion of photoreceptor cell bodies in the outer plexiform layer (arrowheads), and disorganization of horizontal cells lining the top of the outer nuclear layer (asterisks) suggesting defects to the ribbon synapses (40X magnification, scale bars: 20 µm for main panels, 10 µm insets). B : Quantification of synaptophysin intensity in the outer plexiform layer showing a significant reduction in MZKOs (**p = 0.0069). C : Fluorescent staining of transverse cryosections <t>with</t> <t>zn-8</t> and DAPI showing an overall disruption in morphology of the retina with evidence of retinal ganglion cell axon defasciculation in the optic nerves at the chiasm (10X magnification, scale bars: 100 µm for main panels, 20 µm for chiasm insets).
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    Miltenyi Biotec anti cd166 miltenyi biotec
    A : Fluorescent staining of the outer layers of the retina showing comparable zpr1 (Arr3a) staining in the photoreceptor outer segments and pedicles in the outer plexiform layer, but reduced and disrupted synaptophysin (syp) staining, protrusion of photoreceptor cell bodies in the outer plexiform layer (arrowheads), and disorganization of horizontal cells lining the top of the outer nuclear layer (asterisks) suggesting defects to the ribbon synapses (40X magnification, scale bars: 20 µm for main panels, 10 µm insets). B : Quantification of synaptophysin intensity in the outer plexiform layer showing a significant reduction in MZKOs (**p = 0.0069). C : Fluorescent staining of transverse cryosections <t>with</t> <t>zn-8</t> and DAPI showing an overall disruption in morphology of the retina with evidence of retinal ganglion cell axon defasciculation in the optic nerves at the chiasm (10X magnification, scale bars: 100 µm for main panels, 20 µm for chiasm insets).
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    Image Search Results


    A : Fluorescent staining of the outer layers of the retina showing comparable zpr1 (Arr3a) staining in the photoreceptor outer segments and pedicles in the outer plexiform layer, but reduced and disrupted synaptophysin (syp) staining, protrusion of photoreceptor cell bodies in the outer plexiform layer (arrowheads), and disorganization of horizontal cells lining the top of the outer nuclear layer (asterisks) suggesting defects to the ribbon synapses (40X magnification, scale bars: 20 µm for main panels, 10 µm insets). B : Quantification of synaptophysin intensity in the outer plexiform layer showing a significant reduction in MZKOs (**p = 0.0069). C : Fluorescent staining of transverse cryosections with zn-8 and DAPI showing an overall disruption in morphology of the retina with evidence of retinal ganglion cell axon defasciculation in the optic nerves at the chiasm (10X magnification, scale bars: 100 µm for main panels, 20 µm for chiasm insets).

    Journal: PLOS Genetics

    Article Title: Impact of maternal compensation on developmental phenotypes in a zebrafish model of severe congenital muscular dystrophy

    doi: 10.1371/journal.pgen.1011987

    Figure Lengend Snippet: A : Fluorescent staining of the outer layers of the retina showing comparable zpr1 (Arr3a) staining in the photoreceptor outer segments and pedicles in the outer plexiform layer, but reduced and disrupted synaptophysin (syp) staining, protrusion of photoreceptor cell bodies in the outer plexiform layer (arrowheads), and disorganization of horizontal cells lining the top of the outer nuclear layer (asterisks) suggesting defects to the ribbon synapses (40X magnification, scale bars: 20 µm for main panels, 10 µm insets). B : Quantification of synaptophysin intensity in the outer plexiform layer showing a significant reduction in MZKOs (**p = 0.0069). C : Fluorescent staining of transverse cryosections with zn-8 and DAPI showing an overall disruption in morphology of the retina with evidence of retinal ganglion cell axon defasciculation in the optic nerves at the chiasm (10X magnification, scale bars: 100 µm for main panels, 20 µm for chiasm insets).

    Article Snippet: The primary antibodies used in these experiments were 1:200 anti-synaptophysin (Abcam), 1:200 zpr1 (ZIRC), and 1:100 zn-8 (Developmental Studies Hybridoma Bank).

    Techniques: Staining, Disruption